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Progress in Inflammation Research: Pain and Neurogenic Inflammation (1998,...
US $200.00
ApproximatelyRM 845.84
Condition:
Like New
A book in excellent condition. Cover is shiny and undamaged, and the dust jacket is included for hard covers. No missing or damaged pages, no creases or tears, and no underlining/highlighting of text or writing in the margins. May be very minimal identifying marks on the inside cover. Very minimal wear and tear.
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eBay item number:124121608197
Item specifics
- Condition
- ISBN
- 9783764358754
- EAN
- 9783764358754
About this product
Product Identifiers
Publisher
Springer Basel A&G
ISBN-10
3764358750
ISBN-13
9783764358754
eBay Product ID (ePID)
475606
Product Key Features
Number of Pages
Xii, 348 Pages
Language
English
Publication Name
Pain and Neurogenic Inflammation
Subject
Pain Medicine, Neuroscience, General
Publication Year
1998
Type
Textbook
Subject Area
Medical
Series
Progress in Inflammation Research Ser.
Format
Hardcover
Dimensions
Item Weight
53.3 Oz
Item Length
9.3 in
Item Width
6.1 in
Additional Product Features
Intended Audience
Scholarly & Professional
Dewey Edition
21
Number of Volumes
1 vol.
Illustrated
Yes
Dewey Decimal
616/.0473
Table Of Content
A comparative discussion of A? and C fibres in different tissues.- The roles of spinal receptors in nociceptive responses.- Cutaneous hyperalgesia.- Capsaicin and pain mechanisms.- Nitric oxide and inflammatory pain.- Interactions between kinins and the inflammatory pain process.- Sensory neuropeptides, neurogenic inflammation and inflammatory cells.- Inflammatory pain and the joint.- Nerve growth factor and sensory nerve function.- The relationship between hyperalgesia and hyperresponsiveness in the lung.- Neuropeptides and the respiratory tract: Relevance to asthma and rhinitis.- Neuropeptides and the gut: Tachykinins and calcitonin gene-related peptide in intestinal inflammation and pain.- Regulation of meningeal blood flow by neuropeptides: Relevance to migraine.- Sensory peptides: Effects in ageing and wound healing.- Calcitonin gene-related peptides and their receptors.- Tachykinin receptors and the potential of tachykinin antagonists as clinically effective analgesics and anti-inflammatory agents.
Synopsis
Pain and inflammation are inextricably linked phenomena. The observation that chemical mediators with combined pro-inflammatory, algesic and/or hyperalgesic activity occur at the site of inflammation is fundamental not only to our present understanding of the inflammatory process but also to our attempts to devise clini cally useful anti-inflammatory therapies. Over a hundred years ago it was recognised that primary sensory neurones play a crucially important "dual" role in inflammation. By affecting the transfer of infor mation from peripheral nociceptors to the spinal cord, a subpopulation of sensory nerves {"pain fibres"} initiate algesia and hyperalgesia, whose sensations are then modified and fine-tuned in the central nervous system. Equally important is the release from the peripheral terminals of sensory neurones of neuropeptides, the acute effects of which are observed as changes in microvascular tone and perme ability leading to neurogenic inflammation. Over the last decade it has become increasingly clear that this view of the func tion of sensory nerves is somewhat over-simplified. For example, the mechanisms underlying hyperalgesia may, in certain circumstances, be mimicked in other condi tions such as the hypersensivity associated with asthma. Furthermore, it has become increasingly evident that over a longer time period the release of neuropeptides from peripheral sensory nerve endings may also have modulating effects on inmune cells and that this may be relevant to chronic inflammatory disease and possibly also to inflammatory hyperalgesia., Pain and inflammation are inextricably linked phenomena. The observation that chemical mediators with combined pro-inflammatory, algesic and/or hyperalgesic activity occur at the site of inflammation is fundamental not only to our present understanding of the inflammatory process but also to our attempts to devise clini- cally useful anti-inflammatory therapies. Over a hundred years ago it was recognised that primary sensory neurones play a crucially important "dual" role in inflammation. By affecting the transfer of infor- mation from peripheral nociceptors to the spinal cord, a subpopulation of sensory nerves {"pain fibres"} initiate algesia and hyperalgesia, whose sensations are then modified and fine-tuned in the central nervous system. Equally important is the release from the peripheral terminals of sensory neurones of neuropeptides, the acute effects of which are observed as changes in microvascular tone and perme- ability leading to neurogenic inflammation. Over the last decade it has become increasingly clear that this view of the func- tion of sensory nerves is somewhat over-simplified. For example, the mechanisms underlying hyperalgesia may, in certain circumstances, be mimicked in other condi- tions such as the hypersensivity associated with asthma. Furthermore, it has become increasingly evident that over a longer time period the release of neuropeptides from peripheral sensory nerve endings may also have modulating effects on inmune cells and that this may be relevant to chronic inflammatory disease and possibly also to inflammatory hyperalgesia.
LC Classification Number
R1
Item description from the seller
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